PCOS / PCOD Treatment in Gurugram

Polyendocrine Metabolic Ovarian Syndrome, shortened to PMOS. The change was announced on 12 May 2026 after a 14-year international consensus process. Polyendocrine refers to the several hormone systems involved, metabolic to the effects on weight and blood sugar, and ovarian to where the condition shows itself.

Because the old name described the one thing about the condition that is not true. It reduced a complex, lifelong hormonal and metabolic disorder to a misunderstanding about cysts, and the research confirms there is no increase in abnormal cysts on the ovary at all. The name led to missed diagnoses and to treatment that stopped at the ovaries.

No. Your diagnosis has not changed, your treatment does not change, and no new test is needed. If you were told you have PCOS, you have PMOS. It is the same condition under a more accurate name.

An international consensus process led from Monash University, involving more than 50 patient and professional organisations including the Endocrine Society and the AE-PCOS Society, and drawing on over 22,000 survey responses from women with the condition collected in 2017, 2023 and 2025. The final panel vote was 88 of 90 in favour.

No, and it is worth knowing that. Two of the ninety panel members voted against, both patient-advocacy leaders, arguing that it is a very large rebranding effort to change a single letter and that the new name still leaves some questions open. The objection is a reasonable one.

Slowly. There is a three-year transition, with full adoption expected in the 2028 international guideline update. You should expect to keep seeing PCOS on lab forms, prescriptions, hospital records and insurance paperwork for years yet. Both names refer to the same condition.

Not as part of the renaming. Diagnosis still rests on two of three features in an adult. Whether the criteria themselves should change is a separate question that remains under review internationally.

There is none. PCOD is not a recognised medical term anywhere in the world; it is Indian usage that stuck. The idea that PCOD is a milder version and PCOS the serious one was invented in clinics and has no basis in any guideline. If your report says PCOD, it means PCOS.

Not on its own. Polycystic-appearing ovaries on a scan are one of three possible features, and at least two are needed for the diagnosis. Plenty of women have that appearance with entirely normal cycles and normal hormones and do not have the condition.

They are not cysts. They are small follicles that started to grow and never matured, which is exactly what happens when no egg is released. They are not tumours, they do not need removing, they do not turn into anything, and they are unrelated to the ovarian cysts that sometimes need surgery.

Twenty or more in at least one ovary, on modern equipment. Counting fewer than that does not support the diagnosis, and the count alone never makes it.

The appearance often improves once cycles become regular, because follicles are being used rather than accumulating. But the aim of treatment is not to clear the scan picture, and a scan is not the way to judge whether treatment is working.

In an adult, by any two of three: irregular or absent ovulation; excess androgen, either visible as unwanted hair, persistent acne or scalp hair thinning, or measured on a blood test; and polycystic ovaries on a scan or a raised AMH level. Conditions that imitate it, particularly thyroid disease and raised prolactin, are excluded first.

In adults, yes. The 2023 international guideline accepts a raised AMH as an alternative to the ultrasound for the third criterion. This is a genuine change and is not yet widely known. It does not apply to adolescents.

It deserves questioning. Within eight years of a girl's first period, neither the ultrasound nor AMH should be used for diagnosis, because multi-follicular ovaries are simply normal at that age. In adolescents both irregular cycles and androgen excess must be present, and irregular cycles alone in the first year or two after menarche are usually just an immature system settling.

An underactive thyroid, a raised prolactin level, and less commonly adrenal conditions such as non-classical congenital adrenal hyperplasia. All are checked with blood tests. Some are corrected by treating the cause rather than by treating PCOS at all.

Between 10 and 13% of women worldwide using the current criteria. Indian estimates vary widely by region, and a study of over a thousand college students in Delhi NCR found 17.4% - with around three in ten of those cases picked up during the study rather than already known. Under-diagnosis is the norm, not the exception.

Yes, and it is regularly missed for exactly that reason. Insulin resistance is present in lean women with PCOS too. The guideline asks for a lipid profile at diagnosis regardless of age and BMI, which exists precisely so that slim women are not skipped.

Both directions are real, which is why it is so hard to escape. Insulin resistance makes weight easier to gain and harder to lose; extra weight worsens insulin resistance and androgen levels. That is a loop, not a character failing, and it is the reason a modest weight reduction has an effect out of proportion to its size.

Less than people expect. A modest reduction restores spontaneous ovulation in a meaningful proportion of women with PCOS and improves the response to treatment in those who still need it. The target is not a dress size.

Yes. Impaired fasting glucose, impaired glucose tolerance and type 2 diabetes are all commoner, independent of weight. This is why the guideline asks for glucose testing at diagnosis in everyone with the condition, and again every one to three years depending on your other risk factors.

The 75 g oral glucose tolerance test is the most accurate assessment in PCOS - more so than HbA1c alone. It is done at the clinic. A single normal fasting sugar is not a complete answer to this question.

The risk is meaningfully higher. Pooled across 22 studies and 5,507 women with PCOS, 24% developed gestational diabetes. It is a reason to check sugars before conception rather than waiting for the routine test.

There is an increased risk of cardiovascular disease. Nothing about that is inevitable, and it is the reason blood pressure is checked annually and a lipid profile is done at diagnosis rather than when symptoms appear.

It raises the risk of cancer of the uterine lining, because a lining that is never shed keeps thickening. Pooled odds are 2.79 overall and 4.05 in women under 54. Set against an absolute incidence that peaks at roughly 90 cases per 100,000 women a year, that is a reason for regular bleeds rather than for fear - and regular bleeds are easy to arrange.

The principle is that the lining should not be left unshed for long stretches. Cycles that come only every few months, or not at all, are the pattern that needs a plan - usually the pill, cyclical progestogen or a hormonal coil. What is right for you is decided at the consultation, not by a general rule.

Mention it if you snore, wake unrefreshed or are sleepy through the day. Sleep apnoea is significantly commoner in PCOS and, notably, independently of body weight. Treating it improves the metabolic picture as well as the sleep.

It is common enough that the international guideline asks doctors to screen for depression and anxiety in everyone with the condition. Moderate to severe symptoms are frequent. Raising it is not a distraction from the medical conversation; it is part of it.

No, and anyone promising a cure is selling something. It can be controlled very effectively, which is a different and more useful statement. Symptoms return when treatment stops because the underlying condition is still there.

For irregular cycles and for the hair and acne, the combined oral contraceptive pill, with a preference for lower-dose preparations. No particular brand is recommended over another. Where weight is a factor, lifestyle change runs alongside it rather than instead of it.

Cyclical progestogen tablets, or a hormonal IUD, which does the same job for years without a daily tablet and usually makes periods much lighter. The coil is fitted at the clinic as an outpatient procedure. It is the option most women with PCOS are never offered.

It is recommended primarily for the metabolic features rather than as a fertility drug, and it is most useful where insulin resistance is present. The guideline notes it has greater efficacy than inositol. It is not routinely continued into pregnancy.

The international guideline's conclusion is that inositol offers limited clinical benefits in PCOS, and that metformin is more effective. It is heavily marketed in India. It is not harmful; it is simply not the answer it is sold as.

Anti-androgen medication has a limited role and is used where other treatments have not worked or cannot be used, always with reliable contraception alongside because of the risk to a pregnancy. Laser reduces hair effectively in some women. Either way, expect months rather than weeks - hair responds slowly to everything.

Usually, with time. Both respond to lowering androgen levels, and the pill is the usual route. Skin tends to improve before hair, and scalp hair is the slowest of all to recover.

No. It is the commonest cause of ovulatory infertility and it is also among the most treatable. The problem is that eggs are not being released, not that they are absent - in fact the ovary usually holds plenty.

Letrozole is first-line, ahead of clomiphene, and the difference in live births is not small. If tablets do not work the options are metformin alongside them, gonadotropin injections, laparoscopic ovarian drilling, or IVF. Our ovulation induction page sets out the evidence and the monitoring.

Getting glucose, weight where relevant, thyroid and folic acid in order before conception is worth doing and makes the pregnancy safer. It is not a reason to postpone indefinitely, and age still matters more than anything else.

Cycles often become more regular for a while, which is why the myth persists. The condition itself does not go anywhere, and the metabolic side continues to need attention long after your family is complete.

No. All scans are arranged by referral - we do not run imaging at the clinic - and the report and images are then gone through with you here. Consultation, blood tests including the glucose tolerance test, prescribing and follow-up all happen at the clinic.

Consultation, the full blood work-up, prescribing and monitoring of all the medical treatments including ovulation induction, and Mirena insertion and removal as an outpatient procedure. Hysteroscopy, laparoscopy and ovarian drilling are hospital procedures.

Any previous scan reports and blood tests, a rough record of your last several cycle dates, and a list of anything you are already taking, supplements included. If you have a family history of diabetes or thyroid disease, that is worth knowing too.

Yes, and it is the commonest version of this story. Being put on the pill and discharged leaves the metabolic side unexamined for years. A review that checks glucose, lipids and blood pressure is worth booking even when nothing feels wrong.